A ground-breaking, solid, practice-changing, and "dogma-removing" study
I'm confused by studies which conflate all etiologies of pancreatitis.
Assume that in certain populations, ≈ 90% of pancreatitis is due to EtOH and gallstones.
The mechanism of injury is completely different.
Why would we assume a specific intervention would have the same outcome?
I'm confused by studies which conflate all etiologies of pancreatitis.
Assume that in certain populations, ≈ 90% of pancreatitis is due to EtOH and gallstones.
The mechanism of injury is completely different.
Why would we assume a specific intervention would have the same outcome?